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We also suggest studying this relationship using animal models of SSD and ASD to find the molecular pathway affecting the retina-brain axis leading to cognitive impairment, specifically in SSD and ASD

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But, NMN is unable to function as described above in GPX4-knockdown keratinocytes, suggesting that the mechanism of ferroptosis resistance exerted by NMN is still reliant on GPX4 (Feng et al., 2022) (Figure 6)