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Binding to its FGF receptor (FGFR) on hepatocytes, FGF-19 acts as a CYP7A1 repressor, thus contributing to the feed-back inhibition of bile acid synthesis [284,285]
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However, a deeper understanding of the molecular mechanisms leading to APAP-induced liver injury is crucial for developing effective, timely, and targeted therapies to reduce the global burden of APAP toxicity
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