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Niacin attenuated myocyte injury and improved survival during myocardial ischemia and reperfusion [123, 124] and this therapeutic benefit may be associated with the synergistic activation of the glutathione redox cycle, a decrease of the NADH/NAD + ratio and increased glycolysis and lactate efflux, reduction of hydrogen peroxide level, and upregulation of nuclear factor erythroid 2 (Nrf2) related factor [125]
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