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Selegiline also inactivated the 7-EFC O -deethylation activity of CYP2B6 and acted as a mechanism-based inactivation of bupropion metabolism as measured by the decreasing rates of formation of hydroxybupropion with time of incubation as shown in Fig

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Erythrocyte cytochrome b 5 greatly accelerates reduction of methemoglobin by cytochrome b 5 reductase and can also serve as a substrate for hepatic microsomal cytochrome b 5 reductase

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