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This suggests that the Wnt5a/JNK signaling pathway is involved in the endothelial dysfunction of DM ( Recent studies suggest that when Wnt and β-catenin are increased, epidermal cell proliferation, differentiation, and migration are enhanced, and wound healing is accelerated (Yamaguchi et al., 2008) and Wnt protects against vascular endothelial cell damage caused by high glucose levels
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What we DO know, however, is that only 20% (or even less) of the people with symptomatic B12 deficiency have anaemia or altered size of the red blood cells
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