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Therefore, oxidative stress dual markers can potentially be a predictive marker for onset of diabetic mellitus (or even the development of diabetic complications)
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When lysosomal function is severely impaired, lysosome-independent degradation pathways, such as those activated by Bortezomib, a proteasome modulator, can enhance ubiquitination and proteasomal activity, compensating for defective lysosomal function and facilitating the removal of damaged proteins and organelles, thus improving immunotherapy efficacy (133) ( Table 1 )
