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A recent study found that β2-adrenoreceptor (B2AR) agonists (e.g.,: clenbuterol and salbutamol) could reduce α-syn gene expression by modulating transcription through altering histone deacetylase (HDAC) activity at the α-syn gene promoter and enhancer regions, and found that these modifications were neuroprotective in cell line and rodent models (Mittal et al., 2017)