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Semax Rescues Abeta and Cu-Associated Abeta Toxicity It has previously been shown that Aβ 1–42 oligomeric aggregates form insoluble amyloid deposits in the brain and hippocampus and exhibit neurotoxic effects in in vitro and in vivo models, leading to the cognitive dysfunction prevalent in the pathogenesis of AD
GPromo
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Extends telomere length in human cell lines
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