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GHRH receptor of little mice contains a missense mutation in the extracellular domain that disrupts receptor function

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In isolated human hair follicle organ cultures, GHK-Cu at concentrations between 10 and 100 nanomolar extended the anagen phase significantly compared to vehicle controls, an effect accompanied by measurable increases in IGF-1 secretion from dermal papilla cells and reductions in TGF-beta2, the key cytokine that triggers catagen transition [2]