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Interestingly, while mice lacking the HIF1a gene show severe cardiac and vascular malformations leading to embryonic lethality [48], deletion of the hypoxia response elements results in viable offspring with only minor neurological defects in motor neurons [49,50], suggesting that the classical understanding of VEGF transcription may not be the primary means by which its expression is controlled
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All animal procedures were approved by the Institutional Animal Care and Use Committee at the University of Oklahoma Health Sciences Center, and were conducted in compliance with the U.S
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