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Various pathways play a role in ethanol induced tissue injury, including changes in cellular oxidized NAD+, NADH (24), production of acetaldehyde protein adducts (25), induction of CYP2E1 (26), formation of 1-hydroxyethyl free radicals (27), ethanol mediated mitochondrial damage (28), endotoxin derived activation of Kupffer cells and subsequent production of tumour necrosis factor a (29)

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It is plausible to suggest these noted variances may be due to the investigated cell type for IL-6 (astrocytic cell lines, HFAs and monocytes) and IL-1β (astrocytes, MDMs and microglia)